While a number of studies have found that strontium supports
healthy bone mineralization, it is important for people with
impaired renal function to be cautious. It appears that differences
in strontium excretion by impaired kidneys affect strontium
accumulation and alter bone deposition characteristics. Animal
studies have found that high doses of strontium given to rats
with chronic renal failure resulted in bones with fewer osteoblasts
and slower maturing osteoids. Bone formation and mineralization
rates were also slowed. Blood tests showed decreased parathyroid
hormone and increased serum alkaline phosphatase. All effects
were reversed with the withdrawal of strontium.*
2. Cohen-Solal M. Strontium overload and toxicity: impact
on renal osteodystrophy. Nephrol Dial Transplant. 2002;17
Suppl 2:30-4.
3. Oste L et al. Time-evolution and reversibility of strontium-induced
osteomalacia in chronic renal failure rats. Kidney Int. 2005
Mar;67(3):920-30.
For educational purposes only. Consult your physician for any health problems. 
